(C) Quantification of different phenotypes manifesting in MN2a and MN3 after neuronal RNA interference or mis-expression of Ig genes (DIPgamma, DIPkappa, dpr11, dpr1) in comparison to control animals: MN2a axons projecting to the MHE muscle, representing the wild-type (wt) innervation pattern of MN2a (magenta); MN3 axons projecting to the MHD muscle, representing the wild-type (wt) innervation pattern of MN3 (cyan); MN2a or MN3 axons projecting to muscles other than the MHE or MHD termed wiring defects (dark grey); abnormal synaptic morphologies at MN2a or MN3 derived axon terminals termed terminal defects (light grey). Each experiment was performed in triplicates. Note, each genetic experiment was performed in parallel to an adequate control experiment using the same driver line crossed to a line that controls expression of either UAS-RFP or UAS-GFPRNAi. p-values between two genetic conditions were calculated by a two-sided Fisher test. * p < 0.05, **<0.01, *** p < 0.005. 