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Atherosclerosis , the cause of ischemic heart disease , is a condition where the coating of oxidized fats (atheroma) is deposited in the blood vessels . Atherosclerosis is the most common of civilization diseases ( Barac2009mdv ). Oxidation of fat in the blood is proportional to the amount of fatty acids (LDL) in the blood and the amount of free radicals in the blood. The amount of free radicals in turn correlates with the amount of oxygen - therefore, the atheromas are formed only in the arteries. The capture of oxidized fats on the inner surface of the blood vessels (endothelium) invokes macrophages that try to clean up the contaminants by absorbing and degrading oxidized fats. If these oxidized fat particles are too much, the macrophage will eventually die to death, and the pooled lipids will once again pour into the vessel wall. This calls for other macrophages that await the same fate. The atheroma, a kind of "macrophagian cemetery," which has certain characteristics of an inflammatory deposit, gradually develops on the affected site. Cholesterol is accumulated, which is resistant to degradation. Simplified lesson is that atherosclerosis is mainly an immune process and this fact is not mutually exclusive to the cholesterol hypothesis of cardiovascular diseases ( Ignarro2007npa ). Although atherosclerosis (not to interfere with arteriosclerosis ) is the cause of myocardial infarction , it is not one of the direct causes of hypertension ( Shimbo2010edr ). Factors of atherosclerosis Atherosclerosis begins in early adulthood and progresses without causing problems during life. Developed atherosclerosis is untreatable for the time being. The only cure is prevention. Thinking doctors, therefore, try to protect the patients they care about before proceeding. According to current knowledge, atherosclerosis causes two things: - Oxidation of fat in the blood. - Unclear immune pathological process . Oxidation of fat in the blood again requires two things: So we have three parameters to control the development of atherosclerosis: oxygen, blood fat and immunity. Oxygen From the point of view of the amount of oxygen in the blood, I would pragmatically divide the activity into three regimes: (a) sleep, (b) calm, (c) physical exertion. In sleep, the oxygen content in the blood is reduced - the body is saved and repaired at night. At rest, for example, in non-stressful mental work or in light physical activity (walking), the oxygen content is at the basic alert level. In physical exercise mode (physical work, training) the oxygen content is increased according to the intensity of the physical activity. Fat in the blood Roughly speaking, there are fatty droplets of two types: LDL ( low density lipoprotein ) and HDL ( high density lipoprotein ). They are labeled "lipoprotein", but in fact they are just small fats of fat with a little protein, which prevents them from merging. It's like fat in milk, we have less in blood. HDL droplets are "good" (do not cause atherosclerosis) because they are small. They have a high density because they have little fat (fat is light, protein is heavy). LDLs are bad because they are the real, big (and therefore light) creases of fat. They are mainly released from the small intestine into the blood stream. Blood in the vein vein is overcoat after fatty food. The vein vein, as we know, leads to the liver, where it is once again branched into capillaries and where the LDL droplets become the chemical raw material for the hepatic cells, so they should not go too much into the large circulation. But this is likely to occur somewhat, and LDL droplets in the large bloodstream contribute to the development of atherosclerosis. Immune factors of atherosclerosis Growth of atherosclerosis (atherosclerosis) appears to be oxidative stress. But it also causes an inflammatory process that leads to the development of atheroma. It is not clear what is the interaction between oxidation of LDL droplets and the inflammatory process in the vessel wall, but both of these factors are mutually supportive and that both are necessary to develop atherosclerosis ( Hajjar2013bri ). Roughly, LDL droplets are oxidized and trapped on the wall of the arteries (or first trapped and then oxidized). The clutter calls for cleaning macrophages that can not be cleansed, oxidized fat droplets are desired and appear to be filled with foam ("foam cells") under the microscope. These macrophages will then die and the absorbed disorder spills back into the vessel wall. (Oiled fats are what can be found in households on a wall near a frying pan, where they are fried in the pan, and when I clean it, I sometimes feel like I'm going to die of it.) It's going on all the way up to the place where the atherosclerotic bearing mainly cholesterol, which is the most persistent of all fats. We must realize that the development of atherosclerosis must be wrong in this process. It is normal that the vessel wall becomes dirty occasionally and that the macrophages will come clean. There must be some other factor that will cause routine cleaning to fail and create an atheroma. We do not know exactly what this factor is, but it seems that adaptogens that affect the overall "inflammation" (susceptibility of the body to inflammation) and those with a cytoprotective effect may act positively against the onset and progression of atherosclerosis. So how do you protect yourself from atherosclerosis? My short answer to this question is: I do not know. Popular statins and cholesterol lowering seem to go away and cause more or less a side effect than the very cause of atherosclerosis. Popular antioxidants are very important , but our body already knows about them, so if we eat healthy, we do not need to think too much about them. The popular exercise is also not what it used to be. Aerobic overloading, as it turns out, leads to oxidative overload and damage to the body ( Tsai2001odd ), and some "antioxidants" under these conditions turn into pro-oxidants ( Tomasello2012dar ). It gets right the farmer's insight that whosoever has come down, he will be struck. On the other hand, the zero practice is also bad, because most people today (from the point of view of the pramovkaeka) do not practice at all. It is not clear what to advise, apart from the general recommendation of a return to nature and nature. Atherosclerosis and cholesterol Since cholesterol is the most quantitative in atheroma, it has long been suspected of causing atherosclerosis. However, it turns out that atherosclerosis has (as I wrote above) oxidative and immune connections. It is a hypothesis that cholesterol accumulates in atheromas simply because it is most resistant to degradation. Cholesterol malignancy and statin efficacy demonstrated in herbivorous rabbits ( Shen2013eae and many other similar studies) are not automatically related to humans. It seems that cholesterol in diet is far from worrying so much, so butter and eggs are once again healthy foods ( Fernandez2012rdc , Trapani2012rdc ). Statins are important only for some risk groups ( Koh2009ctp ). Atherosclerosis, stress and smoking Even though I think that smoking is now overtaking smoking, it is necessary to say that smoking is proving to be associated with cardiovascular damage and the smoking ban is 100% indicative of all cardiac events. All forms of smoking, including pipes, cigars and marijuana damage the vessels ( Katsiki2013svr ). Damaging effects on the blood vessels include both smoke (tar, carbon monoxide) and nicotine ( Leone2013vpf ). However, I have prepared the floor to say that epidemiological studies on the relationship between smoking and illness do not seem to have the same statistical weight as it seems. I will not quote anything, just say what I think: I would start by saying that patients with schizophrenia are smoking. And I think they smoke because it helps them, and I urge experimenters to explore this hypothesis. I suppose this hypothesis is valid. I also know that people who are prone to stress are also smoking. This could also explain why smokers and lower category workers smoke a lot. It is logical that stress on human psyche is rather negative, so what helps mentally ill can also help mental well-being in stress. We also know that people with poor living conditions suffer more from stress. And - now I come to the conclusion - I think that part of the cardiovascular damage seen in smokers may be the stress of the bad conditions in which the smokers were found and in whom smoking was accustomed. If stress was caused by a poor life situation, part of the vascular damage can be due to cheap food and poor diet. I therefore urge researchers to take into account smoking smoke studies that sometimes smoking is only correlated with a bad life situation that is the cause of health damage. This is a problem that will not solve the nicotine ban. Atherosclerosis, eating and exercise Atherosclerosis, therefore, begins with dirt wall oxidation by oxidized fat LDL droplets, which mainly come from food. Empirically, it is known that the greatest formation of atheromas is in places where the maximum oxygen content is present, whereas in the veins where oxygen is already consumed, the atheromas do not form at all. Another factor is speed and turbulence of the blood stream. Vessels with the highest oxygen content and high flow rates, such as coronary heart arteries, are the first to suffer from atheroma formation. So I should advise that we do not run a marathon with a thin intestine full of fat mutton? Especially when I sometimes run and swim with my stomach? Probably yes. So do not run with a full stomach. But I have to admit that I personally have the impression that my small intestine does not need this advice. I have the impression that my stomach filled with jogging precisely regulates what and how quickly it goes to the small intestine that regulates what, and how quickly, flows into the vein. Or is it advisable to advise on overall saving, less physical activity at all, as some physicians call it today? Or eat less, how has it been recommended for many years to prolong life? Atherosclerosis can be prevented and treated by adaptogens About adaptogens with preventive and curative effects against atherosclerosis write in detail on a more general page on heart disease . A completely exceptional position in the prevention and treatment of cardiovascular problems is ginseng. Protective effect of ginseng from atherosclerosis In terms of scientific medicine, ginseng has long been known for its systemic anti-inflammatory effect and protective effect on blood vessels (mainly via the NO signaling cascade ). The effect directly on atherosclerosis was experimentally demonstrated relatively recently in ginsenoside Rb1 ( He2007peg , Xu2011egr ) and ginsenoside Rd ( Li2011gpc ). Strong indications of a possible effect on atherosclerosis exist with ginsenoside Rg2 ( Cho2013gri ) and compound K ( Lee2011gmc , Park2013cai ). The effect directly on atherosclerosis of coronary arteries was experimentally detected in ginsenoside Rb 1 ( Zhou2005grb ). Other adaptogens with the effect of atherosclerosis - Gnocen notoginseng : Long-term low doses of notoginsengu had a positive effect on blood cholesterol and the amount of LDL fat in rats ( Ji2007hem ) in fatty rats . The specific effect of notoginsengu against atherosclerosis was experimentally detected in rabbits ( Liu2010pns ), rats ( Joo2010isp ) and ApoE knockout mice with genetic susceptibility to atherosclerosis ( Liu2009tpn , Li2011gpc ). - Stools : Show positive results against diabetic atherosclerosis in rats ( Zhang2006msc ). Saline Stasis Salidroside acts against atherosclerosis ( Zhang2012sda ) and cholesterol tissue damage ( Guan2012pes ) in mice. The sacred rosary ( Rhodiola sacra ) has a positive effect on the heart in hypertensive rats ( Yang2013ers ). - It is possible that a Japanese sedative , traditionally used in the treatment of heart disease , also has the effect of atherosclerosis ( Zhang2011mdi ). - Bajkal Shišek: its flavonoid bajkalin and geniposid from jasminic gardenia were active in experimental atherosclerosis in vivo ( Liu2014oab ). I will not continue to enumerate because atherosclerosis is a lifestyle disease, and practically any type of vegetable or healthy food somehow reduces the risk of civilization diseases.
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Ateroskleroze, uzrok ishemijske bolesti srca , je stanje u kojem se prevlaka pohranjena u arterijama oksidiranog masti (ateroma). Ateroskleroza je najčešća od svih bolesti civilizacije ( Barac2009mdv ). Oksidacija lipida u krvi je proporcionalna količine masnih kapi (LDL) u krvi, a količina slobodnih radikala u krvi. Količina slobodnih radikala je pak u vezi sa količinom kisika - dakle ateroma oblika samo u arterijama. Snimanje oksidiranih lipida na unutarnjoj površini krvnih žila (endotel) pozove makrofage koje pokušavaju onečišćenja očistiti oksidirane masti apsorbirati i razgraditi. Ako ti oksidirane čestice masti previše, makrofage na kraju Udrea na smrt i proguta lipidi opet izlije na stijenke krvnih žila. To pozvati dodatne makrofage, koji čeka ista sudbina. Da pogođenih područja postupno stvorio aterom - A „groblje makrofaga”, koji ima određene karakteristike žarište upale. Kolesterol akumulira tamo, što je prilično otporan na degradaciju. Pojednostavljeno pouka je da je uglavnom ateroskleroza imunološki postupak, a ta činjenica ne isključuju kolesterola hipotezom kardiovaskularnih bolesti ( Ignarro2007npa ). Iako je aterotrombot skleroza (ne biti zbunjen ss arterio skleroza ) uzrokuje infarkt miokarda , nije među izravan uzrok hipertenzije ( Shimbo2010edr ). čimbenici ateroskleroze Ateroskleroza počinje u ranoj odrasloj dobi i napreduje tijekom života, bez uzrokovanja problema. Razvijen ateroskleroza je do sada neizlječiva. Jedini lijek je prevencija. Razmišljajući liječnike stoga pokušava pacijenata koje su im važne, prije nego što je postupak za zaštitu. Prema sadašnjim spoznajama, ateroskleroza uzrokuje dvije stvari: - Oksidacija masti u krvi. - Nejasno imuni patološki proces . Oksidacija masti u opet krvi zahtijeva dvije stvari: Oni su, dakle, na raspolaganju su tri parametra kontrolirati razvoj ateroskleroze: kisik, masnoće u krvi i imunitet. kisik U odnosu na količinu kisika u krvi organizma aktivnosti će pragmatički podijeljeni u tri načina: (a) spavanja, (b) stacionarni način (c) režim fizičkog napora. U snu, sadržaj kisika u krvi smanjena - tijelo noću sprema i popravci. Ostatak, kao što je tijekom ne-stresnog mentalni rad ili svjetlo tjelesna aktivnost (hodanje) je sadržaj kisika u osnovnoj budnim razini. Režim fizički napor (fizičkog rada, trening) sadržaja kisika povećava intenzitet fizičke aktivnosti. Masnoće u krvi Grubo govoreći, krvi, masnih kapi su od dvije vrste: LDL (lipoproteina niske gustoće - lipoproteina niske gustoće) i HDL (lipoprotein visoke gustoće - lipoproteina visoke gustoće). Opulently oni se nazivaju „lipoproteina”, ali u stvarnosti oni su samo male kapljice masti s malo proteina, što ih sprečava spajati. To je kao masti u mlijeku, samo krv smo ga manje. HDL kapljice su „dobre” (uzrok ateroskleroza), jer su male. Imaju visoku gustoću, jer postoji samo mala masti (mast je lagan, tvrd proteina). LDL je loše, jer to je pravi, veliki (a time i svjetlo) kapi masti. U krvotok otpuštaju uglavnom iz tankog crijeva. Krv u portalnu venu je visoke masnoće obrok i mliječno. Portalna vena, kao što znamo, dovodi do jetre, gdje se ponovo grane u kapilare i gdje su LDL kapljice postaju kemijski sirovina za stanice jetre, ali ne bi trebala biti prevelika da prođe u promet. No, to se čini donekle ide i LDL kapljice velika cirkulacija doprinosi razvoju ateroskleroze. Imunološki čimbenici za aterosklerozu Počinkem razvoju ateroskleroze naslaga (ateroma) čini do oksidativnog stresa. Ali to također uzrokuje upalni proces koji vodi do stvarnog razvoja ateroma. To nije u potpunosti jasno što je međuigra kapima LDL oksidaciju i upala u stijenke krvnih žila, no poznato je da oba ova dva faktora međusobno učvršćuju i da su oboje potrebni u cilju razvoja ateroskleroze ( Hajjar2013bri ). Otprilike to odvija tako da oksidirani LDL kapljice, i uzima na zidu arterije (ili inicijalno prikuplja i zatim oksidiraju). Nered pozvati svoje poslove makrofaga da čišćenje ne može podnijeti, oksidirani, masnih kapi žudnje, a zatim pod mikroskopom pojavljuje kao puni pjene ( „pjenaste stanice”). Ovi makrofagi zatim umre i apsorbira nered ulije natrag u stijenke krvnih žila. (Zaoxidované masti su ono što se može naći u domovima na zidu u blizini štednjaka, gdje je tava. Kada je dobio očistiti, također ponekad osjećati da iz matrice). Ovo se događa iznova i iznova kako bi lokalno uzgojene aterosklerotskog depozita formirana posebno kolesterola, što je najviše izdržljiva svih masti. Moramo shvatiti da je razvoj ateroskleroze u ovom procesu mora dogoditi nešto loše. Normalno je da je vaskularni zid ponekad prljava i makrofagi koji je došao čist. Mora postojati neki drugi faktor koji uzrokuje rutinu čišćenja ne i stvara aterom. Mi ne znamo što je to faktor, ali čini se da adaptogens utječu na ukupnu „upala” (osjetljivost na upalu u tijelu), a one s citozaštitnog učinka može imati pozitivan učinak protiv stvaranja i napredovanja ateroskleroze. Pa kako se zaštititi od ateroskleroze? To je kratki odgovor na to pitanje je: Ne znam. Popularne statini i smanjuje razinu kolesterola čini se da otići i više raditi na rezultate i nuspojava nego stvarni uzrok ateroskleroze. Popularne antioksidansi su zaista vrlo važna , ali naše tijelo već zna o njima, pa ako jedete zdravo, ne mislimo o njima pretjerano. Popularne vježbe su također nije ono što je nekad. Aerobik preopterećenja, kao što je prikazano, što dovodi do oksidativnog oštećenja i preopterećenja organizma ( Tsai2001odd ) i neke „antioksidansi” pod tim uvjetima se pretvara u pro-oksidansi ( Tomasello2012dar ). Dobivanje istinu iza zajedničkog uvida da onaj tko muči, on je Udrea. S druge strane, nula vježba je također loša, jer većina ljudi danas (u smislu ranog čovjeka) ne vježba uopće. Stoga nije jasno što savjet, osim opće preporuke natrag u prirodu i prirodnost. Ateroskleroza i kolesterol Budući da kolesterol je kvantitativno najviše aterom, ovo dugo se sumnjalo da su ateroskleroza uzroke. Ispada, međutim, da je ateroskleroza (kao što sam napisao gore) oksidacija i imunološki veze. Moguće je hipotezu da kolesterol nakuplja u ateroma jednostavno zato vrlo otporni na razgradnju. Štetnosti kolesterola i učinkovitost statina pokazao na biljoždernih zeca ( Shen2013eae i mnoge druge slične studije) nije automatski vztažitelná na ljude. Čini se da je kolesterol u prehrani do sada ne morate brinuti o izradi maslac i jaja su ponovo zdravi obroci ( Fernandez2012rdc , Trapani2012rdc ). Statini su važni samo u određenim rizičnim skupinama ( Koh2009ctp ). Ateroskleroza, stres i pušenje Iako mislim da je ovo borba protiv pušenja danas pretjerali, potrebno je reći da je pušenje dokazano je povezan s oštećenjem kardiovaskularnog sustava i kako je zabrana pušenja apsolutno indicirana u svim srčanim događajima. Svi oblici pušenja, uključujući cijevi, cigare i marihuana oštećuje krvne žile ( Katsiki2013svr ). Štetan utjecaj na krvne žile ima i dim (katran, ugljični monoksid) i nikotin ( Leone2013vpf ). Ali ja sam spreman reći zemljišta koje epidemiološke studije o povezanosti pušenja i bolesti nemaju statističku težinu kao što se čini. Neću citirati ništa, samo kažem ono što mislim: Ja bih početi se zna da pacijenti s shizofrenija dim. I mislim da su dim, jer im pomaže i tjerati eksperimentalcima istražiti ovu hipotezu. Pretpostavljam da je to hipoteza istinita. Nadalje, znam da je dim i ljudi skloni stresu. To se također može objasniti zašto puno dima zatvorenika i radnika niže kategorije. Logično je da je naglasak na ljudsku psihu djeluje prilično negativno, tako što pomaže mentalno bolestan, može pomoći čak i zdravi psihički pod stresom. Nadalje ističu da sve više ljudi pati od loših životnih uvjeta. A - Sada sam došao do zaključka - Mislim da kao dio štete kardio-vaskularni promatranom kod pušača može eventualno stres sa lošim uvjetima u kojima se nalaze pušača i da pušenje navikli. Ako je stres uzrokovan lošim životnim situacijama, kao dio ozljede krvne žile može biti odgovoran za jeftini obrok i loša prehrana. Dakle, apeliram na istraživača u studijama o štetnosti pušenja su uzeti u obzir mogućnost da su neki pušenje korelira samo s lošim životnim situaciju koja je zapravo uzrok narušavanja zdravlja. To je problem koji zabranjuje nikotin riješen. Ateroskleroza, hrana i vježbanje Ateroskleroza počinje onečišćenja stijenki žila oksidirani LDL kapi masnoće, koje uglavnom dolaze iz hrane. Empirijski je, pak zna da je većina stvaranje ateroma na mjestima gdje je maksimalni sadržaj kisika, dok venama gdje kisik već konzumira, aterom ne čine uopće. Drugi faktor je brzina i turbulencija protoka krvi. Brodovi s najvišim sadržajem kisika i velike brzine protoka, kao što su koronarna arterija srca, dakle prvo da pate stvaranje ateroma. Stoga savjetujem da ne trčanje maraton s tankom crijevu pun ovčjeg masti? Pogotovo kada je on sam ponekad sam trčati i plivati na pun želudac? Vjerojatno da. Dakle, ne trčanje pun želudac. No, moram priznati da se osobno osjećam da mi je tanko crijevo ne treba savjet. Osjećam da napuni želudac dok jogging precizno regulira što i kako brzo pustinja dalje u tanko crijevo, što pak kontrolira što i kako brzo pustinja u portalnu venu. Ili bi trebao savjetovati ukupna štednja, manje fizičke aktivnosti uopće, kao i danas neki liječnici kažu? Ili jesti manje, kao što je preporučeno za mnogo godina produljiti život? Ateroskleroza može spriječiti i liječiti njezine adaptogens O adaptogena sa preventivno i ljekovito djelovanje protiv ateroskleroze pisanja u detalje o općem google bolesti srca . Izvanredna pozicija u prevenciji i liječenju kardiovaskularnih problema ginseng. Zaštitni učinak ginsenga protiv ateroskleroze Sa stajališta znanstvene medicine ginsenga je relativno star njegov sistemskog protuupalno djelovanje i zaštitno djelovanje na krvne žile (uglavnom putem signalnoj kaskadi NO ) .Účinek direktno ateroskleroze eksperimentalno dokazana je relativno nedavno u na ginsenoside Rb 1 ( He2007peg , Xu2011egr ) i ginsenoside Rd ( Li2011gpc ). Snažne indicije moguće djelovanje protiv ateroskleroze postoje samo ginsenoside Rg 2 ( Cho2013gri ) i spoja K ( Lee2011gmc , Park2013cai ). Učinak izravno na ateroskleroze koronarnih arterija je eksperimentalno utvrđeno u ginsenoside Rb 1 ( Zhou2005grb ). Ostali adaptogens s učinkom protiv ateroskleroze - Ginseng notoginseng : Dugotrajna niske doze Notoginseng su štakori s mastima pozitivan učinak na kolesterola i LDL količine masnih čestica u krvi ( Ji2007hem ). Specifična učinak protiv ateroskleroze Notoginseng Utvrđeno je eksperimentalno u zečevima ( Liu2010pns ) štakora ( Joo2010isp ) i ApoE knockout miševa s genetska predispozicija za aterosklerozu ( Liu2009tpn , Li2011gpc ). - Rhodiola : pozitivni rezultati ateroskleroze dijabetičnih štakora ( Zhang2006msc ). Rhodiola Salidrosid djeluje protiv ateroskleroze ( Zhang2012sda ) cholesterolovému i tkiva ( Guan2012pes ) u miševa. Rhodiola sveta (Rhodiola sacra) ima pozitivan učinak na srce u hipertenzivnih štakora ( Yang2013ers ). - Moguće je da sedoulek japanskom , tradicionalno se koristi u liječenju bolesti srca , također ima djelovanje protiv ateroskleroze ( Zhang2011mdi ). - Scutellaria baicalensis : flavonoid i bajkalin geniposid od gardenije jasmina lijeka tretirane s eksperimentalnim ateroskleroze in vivo ( Liu2014oab ). Na popisu neće nastaviti, jer je ateroskleroza bolest stil života i gotovo svake vrste povrća ili zdrave hrane nekako smanjuje rizik od načina života bolesti.