http___www.jinchanhua.cn__p_187_gtlang_hr_1.xml
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Proteinuria treatment Jinchan spend therapy Large the Cordyceps Jinchan flower therapy proteinuria, foamy urine, cloudy urine, chronic kidney disease, hypertension, renal disease, diabetic nephropathy, polycystic kidney disease, renal failure, uremia, protection of residual renal function, prostate disease, urinary frequency, urgency, hematuria The typical symptoms of chronic kidney disease generally 6-8 weeks noticeable effect, the Cicada spend on kidney kidney deficiency caused by tinnitus, osteoporosis, male sexual dysfunction significant effect. The causes of proteinuria and glomerular dysfunction has a close relationship. The main contents are as follows: First, the formation of proteinuria Several physiological proteinuria formation Third, proteinuria, foamy urine harm Fourth, albuminuria cause , Proteinuria What checks , Proteinuria Integrative Medicine Appendix: About the author First, the formation of proteinuria Glomerular endothelial cells from the inner to the outer layer, the base film layer and the epithelial cell layer. This three-cell distribution the filter holes of different sizes and negative charge, so the glomerular capillary barrier function can be divided into two, namely mechanical barrier - filter holes and charge barrier - negative charge. A mechanical barrier - filter holes Glomerular filtration barrier from the inside out by three layers: ① inner layer of capillary endothelial cells. The endothelial cells on the orifice of a diameter of 50-100nm, called fenestrae (Fenestration). Water, various solutes and macromolecular proteins can freely pass through the apertures; but can prevent the blood cells through, play a role in blood cell barrier. ② middle of the base film of the non-cell, was micro-fiber mesh structure. Large molecular substances, such as plasma proteins can not through the base film. The base film is glomerular primary barrier to prevent the filtration of the macromolecular proteins. The ③ outer layer is a glomerular epithelial cells. Epithelial cells with foot processes, the formation of cracks between the foot processes intertwined. Fissures on the layer of filtration slit diaphragm (filtration slit membrane), 4-14nm diameter holes in the membrane, it can prevent from the inside, the two filter out large proteins through the last bulwark filtered. Endothelial cells, basement membrane and the slit diaphragm together constitute the glomerular filtration membrane. Filtration membrane filtration pores of different sizes, only the small molecules to pass easily, and effectively only through the larger pores of larger radius substances, in general, the effective radius of the substance of less than 1.8nm, can be completely filtered. The effective radius of greater than 3.6nm of macromolecules, such as albumin (molecular weight of about 69,000) is almost completely filtration. 2, the charge barrier - negative charge The layers of the filtration membrane contains many negatively charged species, so the permeability of the filtration membrane is determined by the charge of the substance is filtered. These negatively charged substances exclude negatively charged plasma protein, to limit their filtration. While the effective radius of the plasma albumin was 3.5nm, but because of its negative charge, therefore difficult through the filtration membrane. When a variety of pathological damage (including primary and secondary injury) when acting on the kidneys will cause damage to the kidney microcirculation, prompting kidney tissue (functional renal units) ischemia, hypoxia. Hypoxic injury due to ischemia, glomerular capillary endothelial cells. Glomerular capillary endothelial cells, once damaged, will be attracted to the infiltration of inflammatory cells in the blood circulation and the release of pathogenic inflammatory mediators (IL-1, TNF-a, etc.), the pathological damage caused by the loss of kidney inflammation reaction. Kidney in a pathological state, the glomerular basement membrane (GBM), a series of changes occurs: the filtration holes increases or atresia, GBM fracture, the charge barrier damage, kidney permeability enhancement, filtration membrane negatively charged sugar protein is decreased or disappeared, will lead to the negatively charged plasma protein filtration than normal significantly increased. Hence of formed in the clinical proteinuria. Several physiological proteinuria formation Physiological proteinuria?, The name suggests that is not a disease proteinuria. Because some causes urine routine examination of protein a positive reaction. Physiological proteinuria seen in the following circumstances: In ① urine mixed with blood, pus, inflammation or tumor secretions and menstrual blood, vaginal discharge and other conventional the proteinuria qualitative check can be positive. This urine sediment can be seen in the large number of red blood cells, white blood cells and squamous epithelium cells, no casts, urine pelleted by centrifugation or filtration, protein qualitative inspection will significantly reduce or even became negative; ② the urine left for a long period of time or cooling can be precipitated salt crystals, so that the urine was white turbid proteinuria, easily mistaken for heat or add a little acetic acid make turbid urine turned clear, to help you to separate; The ③ urine mixed with semen or prostatic fluid, or secretions under urethritis, urinary protein reaction positive. This case, the patient has the performance of the lower urinary tract or prostate disease, urinary sediment can find sperm, more flat epithelial cells, etc., can make the difference between; ④ lymphatic urine protein containing less, and do not necessarily was chylous like; ⑤ Some drugs, such as rifampicin, Santonin from the urine, the urine can be turbid similar proteinuria, protein qualitative reaction negative. Third, proteinuria, foamy urine harm (1) proteinuria mesangial toxicity: renal failure models, can be observed to the accumulation of serum proteins in glomerular mesangial, these macromolecules in the accumulation of mesangial area can cause damage to the mesangial cells, hyperplasia of various mesangial matrix synthesis, resulting in glomerulosclerosis. Glomerular proteinuria in the nephrotic model, low-density lipoprotein (LDL) and very low density lipoprotein (VLDL) apolipoprotein B, apolipoprotein A deposition of these aggregates eventually lead to glomerulosclerosis. (2) toxic effects of proximal tubule cells: the proteinuria proteinuria, enter the amount of protein in the tubular epithelial cells increased lysosomal activity increased, suggesting that the protein caused by lysosomal spilled into the cytoplasm of the tubule, followed by cell injury can stimulate inflammation and scarring. (3) the proteinuria caused tubule cell biology changes: the emergence of proteinuria kidney disease cell hyperplasia, representing a non-adaptive response, resulting in renal failure. More and more evidence to show that the protein directly conditioning tubule cells, change the growth characteristics of cytokines and matrix proteins phenotypic expression can lead to the tubule basement side release of PDGF, FN and MCP-1-induced fibrosis. (4) proteinuria caused by hypoxia aggravated tubulointerstitial: Proteinuria each extra energy to digest large amounts of protein reabsorption, tubule cells may be caused by hypoxia, hence resulting tubule cell injury Fourth, albuminuria cause (A) renal proteinuria Glomerular proteinuria seen in acute glomerulonephritis, chronic glomerulonephritis, IgA nephritis, occult nephritis. Secondary seen in lupus nephritis and other autoimmune disorders, diabetic nephropathy, purpura nephritis, renal arteriosclerosis and so on. Metabolic disorders proteinuria seen in gout. Divided into two components lesions filtration degree of membrane damage and proteinuria ① selective proteinuria: albumin-based, and a small amount of low molecular weight protein, high molecular weight protein in the urine (IgG, IgA, IgM, C3, C4), semi-quantitative multi + + + + + + +, a typical disease nephrotic syndrome ② non-selective proteinuria: serious damage and fracture of the glomerular capillary wall, there is a high molecular weight protein in the urine, such as immunoglobulins, complement; albumin in molecular weight and low molecular weight protein, urine protein, immunoglobulin protein / albumin ratio> 0.5, semi-quantitative + + + + + quantitative between 0.5 ~ 3.0g/24h, more common in primary glomerular diseases, such as rapidly progressive glomerulonephritis, chronic nephritis, membranous or membrane glomerulonephritis and secondary glomerular diseases, such as diabetes, nephritis, lupus nephritis. Non-selective proteinuria poor prognosis. Strenuous exercise, the long march, the high temperature environment, fever, cold environment, mental stress, congestive heart failure and proteinuria. Tubular proteinuria, the most common variety of causes of interstitial nephritis, renal vein thrombosis, renal artery embolization, heavy metal salts poisoning. Kidney organization proteinuria, also known as secretory proteinuria. Due to the process of the formation of urine, the protein produced by the tubular metabolism penetrate due to the urine. (B) non-renal proteinuria Humoral proteinuria, also known as overflow proteinuria, such as multiple myeloma. Organizational proteinuria, such as malignant urine protein, viral infections generated host proteins like. Lower urinary tract protein mixed with urine cause proteinuria seen in urinary tract infections, urinary tract epithelial cell shedding mucin secretion and urinary tract. , Proteinuria do what? (A) history History of such as edema, hypertension happens, history of diabetes, allergic purpura history, history of drug use damage the kidneys, the history of heavy metal salts poisoning, proteinuria, and the history of connective tissue disease, metabolic disease and gout episode. (B) the medical examination of proteinuria Note edema and serous effusions, bone and joint inspection, the degree of anemia, and heart, liver, kidney and signs checks. Fundus examination the acute nephritis the fundus normal or mildly vasospasm, chronic nephritis fundus arteriosclerosis, bleeding, oozing, diabetic nephropathy often diabetic fundus. (C) laboratory tests of proteinuria Urine protein checks can be divided into qualitative and quantitative tests and special inspections. Qualitative checks, preferably morning urine, morning urine is the most concentrated, and may exclude orthostatic proteinuria. The qualitative check the only screening test is not as accurate urinary protein content indicators. Urinary protein excretion check Special examination of the urine protein, urine protein electrophoresis can distinguish selective proteinuria and non-selective proteinuria. Multiple myeloma urine protein electrophoresis typing. Diagnosis of early renal tubular dysfunction measured by radioimmunoassay more helpful. Proteinuria in Integrative Medicine Nephrology, Longhua Hospital Affiliated to Shanghai University of Traditional Chinese Medicine, Hong Kong University of Science and Technology, Zhejiang University of Traditional Chinese Medicine, Suzhou Institute of Pharmaceutical, Shanghai Pan Asia Life Sciences Research Institute to carry forward the national treasures of Chinese medicine the Cordyceps Jinchan spend their responsibility, scientific research and discovery opened its mysterious veil gradually by the people know. 1, kidney disease, male sexual dysfunction, national key disciplines Longhua Hospital, Department of Nephrology, using the Yunnan Wild Jinchan flower therapy proteinuria, foamy urine, cloudy urine, chronic kidney disease, diabetic nephropathy, hypertensive nephropathy, renal failure, uremia, residual protection renal, prostate disease, urinary frequency, urgency, hematuria and kidney of tinnitus caused by kidney deficiency, osteoporosis, male sexual dysfunction, usually 4-8 weeks to obtain a significant effect. 2, intractable insomnia, chronic insomnia, habitual insomnia rational drug use Jinchan generally 2-4 weeks spent significant effect. 3, tumor, Japan, Hong Kong University of Science and Technology, Zhejiang University, Suzhou University, Institute of Traditional Chinese Medicine and other studies confirm cancer after surgery, pancreatic cancer, liver cancer, lung cancer, breast cancer, stomach cancer, bladder cancer, advanced cancer, anti-relapse Anti transfer efficacy prominent. Rapid elimination of tumor radiotherapy and chemotherapy toxicity, such as nausea, vomiting, weakness, hair loss, decreased blood cell, insomnia, enhance physical fitness. 4, Japan is mainly used in the field of diabetes, cancer and other diseases. Mainly used in low immunity, fatigue, sub-health, longevity groups. (Proteinuria Integrative Medicine mainly taken from the part of community health, "Xinmin Evening News" special edition, 2012-07-28)
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Proteinurija Jinchan liječenje provesti terapiju Veliki cordyceps Jinchan cvijet terapija proteinurija, pjenast urin, oblačno urina, kronična bolest bubrega, hipertenzija, bolest bubrega, dijabetička nefropatija, policistična bolest bubrega, insuficijencije bubrega, uremija, zaštita rezidualne funkcije bubrega, bolesti prostate, mokraćnog frekvencija, hitnost, hematurija Tipični simptomi kronične bolesti bubrega općenito 6-8 tjedna vidljiv učinak, cvrčak potrošiti na bubrega bubrega nedostatak uzrokovane tinitus, osteoporoze, muškarac seksualne disfunkcije značajan učinak. Uzroci proteinurija i glomerularna disfunkcije ima blizak odnos. Glavni sadržaji su kako slijedi: Prvo, formiranje proteinurije Nekoliko fiziološki proteinurija formiranje Treće, proteinurija, pjenast urina štete Četvrto, albuminuriju uzrok , Proteinurija Što provjerava , Proteinurija Integrativna medicina Dodatak: O autoru Prvo, formiranje proteinurije Glomerula endotelne stanice iz unutarnjeg na vanjski sloj, sloj bazne filma i epitelnih stanica sloj. Ova tri-stanica distribuciju filter rupa različitih veličina i negativnog naboja, tako glomerularne kapilarni zaštitna funkcija može se podijeliti na dva, odnosno mehanička barijera - filter rupe i naknada barijera - negativni naboj. Mehanička barijera - filter rupe Glomerularne filtracije barijere iznutra prema tri sloja: ① unutarnji sloj kapilarnih endotelnih stanica. U endotelne stanice na otvora promjera 50-100nm, zove fenestrae (fenestracije). Voda, razne otopljenim tvarima i makromolekularni proteini mogu slobodno proći kroz otvore, ali može spriječiti krvne stanice kroz, igraju ulogu u krvnih stanica barijere. ② sredina osnovnog filma non-stanici, bio je mikro-vlakana mrežaste strukture. Veliki molekularni tvari, kao što su plazma proteina ne mogu preko osnovnog filma. Baza film je glomerularna primarna barijera kako bi se spriječilo filtraciju makromolekula proteina. ③ vanjski sloj je glomerula epitelne stanice. Epitelne stanice sa stopala procesa, stvaranje pukotina između stopala procesa isprepletenih. Pukotina na sloj filtracije slit dijafragme (dijafragma filtracija membrana), 4-14nm promjer rupe u membrani, to može spriječiti iznutra, dvije filtrirati velikih proteina kroz posljednje predziđu filtrira. Endotelnim stanicama, bazalne membrane i razrez dijafragma zajedno čine membranu glomerularne filtracije. Filtracija membranske filtracije pore različitim veličinama, samo malih molekula da prođe lako i učinkovito samo kroz veće pore većih radijusa tvari, u cjelini, na snazi radijus tvari manje od 1.8nm, može biti potpuno filtrira. Efektivni polumjer veći od 3.6nm makromolekula, kao što su albumin (molekularna masa od oko 69.000) gotovo je u potpunosti filtriranje. 2, naboj barijera - negativni naboj Slojevi filtracije membrane sadrži mnoge negativno nabijene vrste, tako da je propusnost filtracije membrane određuje zadužen tvar se filtrira. Ove negativno nabijene tvari negativno isključuju naplaćuje plazma protein, kako bi ograničili svoju filtraciju. Dok efektivni polumjer plazme albumin bio 3.5nm, ali zbog njegovog negativnog naboja, stoga teško kroz filtracije membrana. Kada razne patološke štete (uključujući i primarnog i sekundarnog ozljede) kada djeluje na bubrege će dovesti do oštećenja bubrega mikrocirkulacije, što je navelo bubrega tkiva (funkcionalni bubrega jedinice) ishemiju, hipoksiju. Hipoksična ozljede zbog ishemije glomerularne kapilarne endotelne stanice. Glomerularne kapilarne endotelne stanice, jednom oštećena, bit će privlačiti infiltracije upalnih stanica u krvotok i oslobađanje od patogenih upalnih medijatora (IL-1, TNF-a, itd.), patoloških štete uzrokovane gubitak bubrega upala reakcije. Bubreg u patološkom stanju, membrana glomerularna podrum (GBM), niz promjena događa: za filtriranje rupe povećava ili atresia, GBM lom, šteta naplatiti barijera, bubrega propusnost pojašnjenje, filtracija membrana negativno nabijen šećer protein je smanjena ili nestala, da će dovesti do negativno nabijeni plazma protein filtracije od normalne značajno povećane. Stoga od formirana u kliničkoj proteinurije. Nekoliko fiziološki proteinurija formiranje Fiziološka proteinurija?, Ime sugerira da nije bolest proteinurija. Budući da su neki rutinski uzroci urina ispitivanje proteina pozitivna reakcija. Fiziološka proteinurija vidjeti u sljedećim okolnostima: U ① urina pomiješanog s krvlju, gnoj, upala ili tumor sekreta i menstrualna krv, vaginalni iscjedak i druge konvencionalne proteinurija kvalitativna provjera može biti pozitivan. Ovo sedimenta urina može se vidjeti u velikom broju crvenih krvnih stanica, bijelih krvnih stanica i stanica pločastog epitela, bez postava, urina peletirana centrifugiranjem ili filtracije, proteina inspekcija kvalitativni će značajno smanjiti ili čak postala negativna; ② urina lijevo za dugo vremensko razdoblje ili hlađenje može biti precipitated kristali soli, tako da urin bio bijelo mutna proteinurija, lako zamijeniti za vrućine ili dodati malo octene kiseline čine mutan urin okrenuo jasno, koji će vam pomoći da odvojiti; ③ urin pomiješan s sjemena ili prostate tekućine, ili izlučivanjem pod uretritisa, mokraćnog proteina reakcije pozitivne. Ovaj slučaj, pacijent ima izvedbu donjeg urinarnog trakta ili bolesti prostate, mokraćnog sedimenta možete pronaći spermu, više ravne epitelne stanice, itd., mogu napraviti razliku između; ④ limfna urina proteina sadrže manje, i ne moraju nužno je chylous poput; ⑤ Neki lijekovi, poput rifampicina, Santonin iz urina, urin može zamuti sličan proteinurija, proteina kvalitativni reakcije negativne. Treće, proteinurija, pjenast urina štete (1) proteinurija mesangial toksičnost: zatajenjem bubrega modeli, može se primijetiti da akumulacije serumskih proteina u glomerularne mesangial, te makromolekule u akumulaciji mesangial području može dovesti do oštećenja stanica, mesangial hiperplazija razne mesangial matrica sinteza, što je rezultiralo u glomeruloskleroza. Glomerularne proteinurija u nefrotički modela, low-density lipoprotein (LDL) i lipoproteina vrlo niske gustoće (VLDL) apolipoproteina B, apolipoproteina taloženje tih agregata na kraju dovesti do glomeruloskleroza. (2) toksični učinci proksimalni tubuli stanica: proteinurija proteinurija, unesite količinu proteina u cjevaste epitelnih stanica povećao lizosomskog aktivnost porasla, sugerirajući da proteina uzrokovane lizosomskog izlilo se u citoplazmi na tubulu, nakon čega slijedi stanica ozljede mogu potaknuti upalu i ožiljke. (3) proteinurija izazvao cjevčica promjene stanične biologije: Pojava proteinurija bolesti bubrega stanica hiperplazija, što predstavlja ne-adaptivni odgovor, što je rezultiralo zatajenjem bubrega. Sve više i više dokaza koji pokazuju da su proteini izravno uređaj cjevčica stanice, promijeniti rasta karakteristike citokina i matrice proteina fenotipske ekspresije može dovesti do bočnog cjevčica podrumu izdanju PDGF, FN i MCP-1-inducirane fibroze. (4) proteinurija uzrokovane hipoksijom teške Tubulointersticijski: Proteinurija svaka dodatna energija za probaviti velike količine proteina reapsorpcije, cjevčica stanice mogu biti uzrokovane hipoksijom, stoga što je rezultiralo cjevčica stanica ozljeda Četvrto, albuminuriju uzrok () Bubrežne proteinurija Glomerularne proteinurija vidjeti u akutnog glomerulonefritisa, kronični glomerulonefritis, IgA nefritis, okultne nefritis. Sekundarna vidjeti u lupus nefritis i drugih autoimunih bolesti, dijabetička nefropatija, purpura nefritis, bubrega arterioskleroze i tako dalje. Metabolički poremećaji proteinurija vidjeti u gihta. Podijeljena je na dvije komponente lezija filtracije stupanj oštećenja membrane i proteinurijom ① selektivna proteinurija: albumina-based, i mali iznos niske molekularne težine proteina, visoke molekularne težine bjelančevina u mokraći (IgG, IgA, IgM, C3, C4), polu-kvantitativno više + + + + + + +, tipična bolest nefrotskog sindroma ② neselektivni proteinurija: ozbiljna oštećenja i prijelom glomerularne kapilarne zid, postoji visoka molekularna težina bjelančevina u urinu, kao što su imunoglobulini, nadopuniti; albumina u molekularnoj težini i niske molekularne težine bjelančevina, urina proteina, Imunoglobulina proteina / albumina omjer> 0,5, polu-kvantitativno + + + + + kvantitativna između 0,5 ~ 3.0g/24h, češći u osnovnim glomerularne bolesti, kao što su brzo progresivni glomerulonefritis, kronični nefritis, Membranozni ili membrana glomerulonefritis i sekundarni glomerula bolesti, kao što su dijabetes, nefritis, lupus nefritis. Non-selektivna proteinurija loše prognoze. Naporan vježba, dugo ožujak, visoka temperatura okoline, temperatura, hladno okruženje, psihički stres, kongestivnog zatajenja srca i proteinurija. Tubular proteinurija, najčešći izbor uzroka intersticijski nefritis, bubrega venska tromboza, bubrežne arterije embolizacijom, heavy metal soli trovanja. Bubreg organizacija proteinurija, također poznat kao sekretorne proteinurije. Zbog procesu formiranja urina, protein koji proizvodi cjevaste metabolizma prodrijeti zbog urina. (B) ne-renalne proteinurija Humoralne proteinurija, također poznat kao preljeva proteinurije, kao što su multipli mijelom. Organizacijski proteinurija, kao što su maligne urina proteina, virusne infekcije generira niz proteini sviđa. Donja urinarnog trakta proteinski miješati s urina uzrok proteinurije vidio u uroinfekcija, urinarnog trakta epitelnih stanica prolijevanja mucinom izlučivanje i urinarnog trakta. , Proteinurija učiniti što? (A) Povijest Povijest kao što su edem, hipertenzija dogodi, povijest dijabetesa, alergijske purpura povijest, povijest korištenja droga oštećenja bubrega, povijest heavy metal soli trovanja, proteinurije, i povijesti vezivnog tkiva bolesti, metaboličke bolesti i gihta epizode. (B) liječnički pregled proteinurije Napomena edem i serumske izljeva, kostiju i zglobova inspekcije, stupanj anemije, i srce, jetra, bubreg i znakove čekove. Fundus ispit akutne nefritis fundusa normalan ili blago vazospazam, kronični nefritis fundusa arterioskleroze, krvarenje, curenje, dijabetička nefropatija često dijabetes fundusa. (C) laboratorijske testove proteinurije Urina proteina provjere mogu se podijeliti u kvalitativnim i kvantitativnim testovima i posebne inspekcije. Kvalitativni provjere, po mogućnosti jutarnji urin, jutarnji urin je najviše koncentriran, i može izuzimati ortostatsku proteinurije. Kvalitativno provjeriti samo test probira nije kao točne mokraćnih sadržaj proteina pokazatelja. Mokraćni lučenje proteina provjera Posebna pregled urina proteina, urin elektroforeza proteina može razlikovati selektivne proteinurije i neselektivnom Proteinurija. Multipli mijelom urina elektroforeza proteina tipkanje. Dijagnoza ranog renalna tubularna disfunkcije mjeri radioimmunoassay više korisnih. Proteinurija u integrativnu medicinu Nefrologiju, Longhua bolnica Povezana Šangaj Sveučilišta tradicionalne kineske medicine, Hong Kong Sveučilišta znanosti i tehnologije, Zhejiang University tradicionalne kineske medicine, Suzhou Institut Pharmaceutical, Šangaj Pan Azija Life Sciences Research Institute nositi naprijed nacionalnih blaga kineske medicine cordyceps Jinchan provesti svoju odgovornost, znanstvena istraživanja i otkrića otvorila njegov tajanstveni veo postupno od strane ljudi znaju. 1, bolesti bubrega, muškarac seksualne disfunkcije, nacionalni ključ discipline Longhua bolnica, Zavod za nefrologiju, koristite Yunnan Wild Jinchan cvijet terapija proteinurija, pjenast urin, maglovitih urin, kronične bolesti bubrega, dijabetička nefropatija, hipertenzivna nefropatija, zatajenje bubrega, uremija, zaostala zaštite bubrega, bolesti prostate, mokraćnog frekvencija, hitnost, hematurija i bubrega tinnitus uzrokovan bubrega nedostatak, osteoporoze, muške seksualne disfunkcije, obično 4-8 tjedan dobiti značajan utjecaj. 2, tvrdoglav nesanica, kronične nesanice, nesanica uobičajeno racionalno korištenje droga Jinchan općenito dva do četiri tjedan proveo značajan učinak. 3, tumora, Japan, Hong Kong Sveučilišta znanosti i tehnologije, Zhejiang University, Suzhou Sveučilište, Institut za tradicionalne kineske medicine i drugih studija potvrđuju raka nakon operacije, rak gušterače, jetre, rak pluća, rak dojke, rak želuca, rak mokraćnog mjehura, napredni raka, anti-Relapse Anti prijenos uspješnosti istaknuti. Automatsko uklanjanje tumora radioterapijom i kemoterapijom toksičnost, kao što su mučnina, povraćanje, slabost, gubitak kose, smanjena krvnih stanica, nesanice, poboljšati fizičku kondiciju. 4, Japan se uglavnom koristi u području dijabetesa, raka i drugih bolesti. Uglavnom se koristi u niskim imunitet, umor, pod-zdravlje, dugovječnost grupe. (Proteinurija Integrativna medicina uglavnom preuzet iz dijela zdravlja zajednice, "Xinmin Evening News" Special Edition, 2012/07/28)