  More virulent strains of influenza virus subtypes H1N1 appeared in 2007 and
H3N2 in 2011. The amino acid differences from prior less virulent strains
appear to be small when tabulated through sequence alignments and counting site
identities and similarities. Here we show how analyzing fractal hydropathic
forces responsible for globular compaction and modularity quantifies the
mutational origins of increased virulence, and also analyzes receptor sites and
N-linked glycan accretion.
