  When the body is infected, it mounts an acute inflammatory response to rid
itself of the pathogens and restore health. Uncontrolled acute inflammation due
to infection is defined clinically as Sepsis and can culminate in organ failure
and death. We consider a three dimensional ordinary differential equation model
of inflammation consisting of a pathogen, and two inflammatory mediators. The
model reproduces the healthy outcome and diverse negative outcomes, depending
on initial conditions and parameters.when key parameters are changed and
suggest various therapeutic strategies. We suggest that the clinical condition
of sepsis can arise from several distinct physiological states, each of which
requires a different treatment approach. We analyze the various bifurcations
between the different outcomes
